PHYSIOLOGICAL EFFECTS OF PULMONARY HYPERTENSION ON CARDIAC FUNCTION
The right and left sides of the heart are functionally integrated by their anatomical contiguity. There is continuity between their free walls, they share a common wall (the intraventricular septum), and they are covered by the pericardium. When pulmonary vascular resistance is normal, the right ventricle serves as a capacitance chamber, performing only minimal contractile work. It compensates ineffectually for acute rises in pulmonary artery pressure, and acutely it can only generate a mean pressure of 40 mm Hg. Acute elevations of right ventricular pressures also interfere with left ventricular performance, presumably owing to a shift in the intraventricular septum to the left. Chronic elevations of pulmonary artery pressure cause gradual hypertrophy of the right ventricle, which eventually allows it to generate pressures equal to those in the left ventricle.
- EMBOLIC DISEASE
- OBLITERATIVE OR OBSTRUCTIVE PULMONARY HYPERTENSION
- CAUSES OF PULMONARY HYPERTENSION
- Idiopathic Pulmonary Fibrosis
- HYPERKINETIC PULMONARY HYPERTENSION
- TREATMENT
- DIFFUSE INFILTRATIVE DISEASES OF THE LUNG
- Miscellaneous
- Pulmonary Infiltrates with Eosinophilia PIE
- POSTCAPILLARY PULMONARY HYPERTENSION
- CLINICAL FEATURES OF PULMONARY HYPERTENSION
- Pulmonary Vasculitis
- CLINICAL MANIFESTATIONS
- SPECIFIC ENTITIES - DISEASES WITH KFiOWIi ETIOLOGIES -
- Pulmonary Hemorrhagic Disorders
- DIFFUSE LUNG DISEASE OF UNKNOWN ETIOLOGY
- Hypersensitivity Pneumonitis
- EFFECTS OF PULMONARY HYPERTENSION ON PULMONARY FUNCTION
- Other Clearly Extrinsic Causes of Diffuse Infiltrative Lung Disease
- Sarcoidosis
- PHYSIOLOGICAL EFFECTS OF PULMONARY HYPERTENSION ON CARDIAC FUNCTION